| dc.contributor.author |
Karatzi, K |
en |
| dc.contributor.author |
Papamichael, C |
en |
| dc.contributor.author |
Karatzis, E |
en |
| dc.contributor.author |
Papaioannou, TG |
en |
| dc.contributor.author |
Stamatelopoulos, K |
en |
| dc.contributor.author |
Zakopoulos, NA |
en |
| dc.contributor.author |
Zampelas, A |
en |
| dc.contributor.author |
Lekakis, J |
en |
| dc.date.accessioned |
2014-06-06T06:47:35Z |
|
| dc.date.available |
2014-06-06T06:47:35Z |
|
| dc.date.issued |
2007 |
en |
| dc.identifier.issn |
01675273 |
en |
| dc.identifier.uri |
http://dx.doi.org/10.1016/j.ijcard.2006.07.200 |
en |
| dc.identifier.uri |
http://62.217.125.90/xmlui/handle/123456789/3697 |
|
| dc.subject |
Cigarette smoking |
en |
| dc.subject |
Endothelial dysfunction |
en |
| dc.subject |
FMD |
en |
| dc.subject.other |
cigarette smoke |
en |
| dc.subject.other |
adult |
en |
| dc.subject.other |
artery blood flow |
en |
| dc.subject.other |
artery diameter |
en |
| dc.subject.other |
artery dilatation |
en |
| dc.subject.other |
article |
en |
| dc.subject.other |
B scan |
en |
| dc.subject.other |
cigarette smoking |
en |
| dc.subject.other |
clinical article |
en |
| dc.subject.other |
controlled study |
en |
| dc.subject.other |
endothelial dysfunction |
en |
| dc.subject.other |
human |
en |
| dc.subject.other |
priority journal |
en |
| dc.subject.other |
time |
en |
| dc.subject.other |
Adult |
en |
| dc.subject.other |
Brachial Artery |
en |
| dc.subject.other |
Endothelium, Vascular |
en |
| dc.subject.other |
Humans |
en |
| dc.subject.other |
Regional Blood Flow |
en |
| dc.subject.other |
Smoking |
en |
| dc.subject.other |
Time Factors |
en |
| dc.subject.other |
Vasodilation |
en |
| dc.title |
Acute smoke-induced endothelial dysfunction is more prolonged in smokers than in non-smokers |
en |
| heal.type |
journalArticle |
en |
| heal.identifier.primary |
10.1016/j.ijcard.2006.07.200 |
en |
| heal.publicationDate |
2007 |
en |
| heal.abstract |
Background: The aim of the study was to compare acute endothelial dysfunction caused by smoking one cigarette between smokers and non-smokers. Methods: Thirty volunteers, 15 smokers and 15 non-smokers, were asked to smoke the same type of cigarette and endothelial function was assessed by FMD (flow mediated dilatation) at fast, 30, 60 and 90 min post-smoking. Results: Overall response of FMD after smoking was significantly different between smokers and non-smokers (p = 0.011). Endothelial dysfunction after smoking remained significant for 1-h in smokers (p = 0.002), but only for 30 min in non-smokers (p = < 0.001). FMD 1-h after smoking was significantly higher in non-smokers than in smokers (p = 0.002). Conclusions: Smokers seem to have sustained and more intensive unfavourable effects in endothelial function than non-smokers after smoking one cigarette. It is possible that long-term smoking diminishes the capacity of endothelium to counterbalance external factors that cause endothelial dysfunction. © 2006 Elsevier Ireland Ltd. All rights reserved. |
en |
| heal.journalName |
International Journal of Cardiology |
en |
| dc.identifier.issue |
3 |
en |
| dc.identifier.volume |
120 |
en |
| dc.identifier.doi |
10.1016/j.ijcard.2006.07.200 |
en |
| dc.identifier.spage |
404 |
en |
| dc.identifier.epage |
406 |
en |